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Amyloidosis (AL),
also known as light-chain amyloidosis, is a rare plasma cell disorder in
which abnormal immunoglobulin light chains produced by the bone marrow
misfold and form amyloid deposits. These deposits accumulate in vital
organs such as the heart, kidneys, liver, nerves, and gastrointestinal
tract, interfering with their normal function. The condition is often
associated with plasma cell disorders like multiple myeloma or monoclonal
gammopathy.
Symptoms vary depending on
the organs involved and may include fatigue, swelling of the legs, unexplained weight
loss, shortness of breath, numbness or tingling in the hands and feet,
enlarged tongue, and protein loss in the urine. Because the symptoms are often
nonspecific, diagnosis can be delayed. Confirmation typically involves blood
and urine tests, imaging studies, bone marrow examination, and tissue biopsy
demonstrating amyloid deposits.
Treatment aims to reduce the
production of abnormal light chains using therapies similar to those used for multiple
myeloma, including chemotherapy, targeted medications, and, in
selected patients, autologous stem cell transplantation. Supportive care is
essential to manage organ-related complications. Early diagnosis and timely
treatment significantly improve outcomes, helping preserve organ function and
enhance quality of life for individuals living with AL amyloidosis.