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Thalassaemia is
a group of inherited blood disorders characterised by reduced or absent
production of haemoglobin, the protein in red blood cells responsible for
carrying oxygen throughout the body. The condition occurs due to genetic
mutations affecting the alpha or beta globin chains of haemoglobin,
resulting in alpha-thalassaemia or beta-thalassaemia. Depending
on the severity, symptoms may range from mild anaemia to
life-threatening complications.
Individuals with thalassaemia
commonly experience fatigue, weakness, pale skin, shortness of breath,
dizziness, delayed growth, poor appetite, and enlarged spleen or liver. Severe
forms, such as thalassaemia major, often develop during infancy and
require regular blood transfusions to maintain healthy haemoglobin
levels. Repeated transfusions may lead to iron overload, which can damage the
heart, liver, and endocrine glands if not properly managed.
Diagnosis involves complete blood count (CBC), peripheral blood smear, haemoglobin electrophoresis,
iron studies, and genetic testing. Treatment depends on disease severity
and may include folic acid supplements, regular blood transfusions, iron
chelation therapy to remove excess iron, and management of associated
complications. In selected patients, stem cell or bone marrow transplantation may provide a potential cure. Genetic counselling and
carrier screening are important for families at risk. With early diagnosis,
appropriate treatment, and regular follow-up, many people with thalassaemia can
achieve improved health and quality of life.