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Hypogonadism is
an endocrine disorder in which the gonads—testes in males and ovaries in
females—fail to produce adequate sex hormones such as testosterone
or estrogen. It may arise before, during, or after puberty and is
broadly classified into primary (gonadal failure) and central or
secondary (hypothalamic–pituitary dysfunction) types. Common causes include
genetic syndromes (like Klinefelter or Turner syndrome), autoimmune
diseases, infections, tumors, radiation, surgery, or chronic illnesses
affecting the gonads or pituitary gland.
In children, hypogonadism
typically presents with delayed or absent puberty, poor growth, lack of breast
development in girls, and deficient genital or secondary sexual character
development in boys. Adults may notice amenorrhea, hot flashes,
low libido, erectile dysfunction, loss of body hair, reduced muscle mass,
fatigue, and mood changes. Diagnosis involves detailed history, physical
examination, and blood tests to measure sex hormones, LH, FSH, and other
pituitary hormones, sometimes supported by imaging such as MRI or ultrasound.
Treatment usually centers on
hormone replacement therapy—testosterone for males and estrogen
with or without progesterone for females—to restore normal puberty, sexual
function, and bone health, while addressing the underlying cause when
possible. With timely and appropriate management, most patients can achieve
improved quality of life and, in select cases, restored fertility.