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Hypocalcemia occurs
when serum calcium levels fall below the typical range of 8.5 to 10.2
mg/dL. Because calcium is a vital electrolyte, its deficiency
disrupts critical physiological processes, including bone mineralization,
blood clotting, and enzymatic activity. The condition often arises
secondary to underlying health issues such as hormonal imbalances—notably
parathyroid deficiency, vitamin D insufficiency, hypomagnesemia, or
chronic kidney disease.
Mild hypocalcemia may
remain asymptomatic, especially if the decline is gradual. However, as
levels drop further, the increased excitability of nerves and muscles can lead
to the mnemonic "CATs go numb"—representing convulsions, arrhythmias,
tetany, and paresthesia. Symptoms often manifest as tingling in the
extremities and around the mouth, involuntary muscle spasms or cramps, and in
severe cases, life-threatening seizures or cardiac rhythm disturbances.
Diagnosis and Management
Diagnosis is established
through blood tests measuring serum calcium, often accompanied by evaluations
of PTH, vitamin D, phosphate, and magnesium levels to identify
the root cause. Treatment strategies focus on correcting the primary
underlying condition while restoring systemic calcium balance. Common
interventions include oral calcium and vitamin D supplementation
for chronic management, while acute or severe manifestations may require
hospitalization and the administration of intravenous calcium to rapidly
stabilize neurological and cardiac function.